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162 articles — page 6 of 14
Baricitinib is an oral JAK1/2 inhibitor approved for alopecia areata and rheumatoid arthritis with emerging Phase 3 data for vitiligo. Here is what the trial evidence shows and how it compares to other JAK inhibitors.
Self-tanners work differently on depigmented vitiligo patches than on normal skin — the chemistry explains why, and sets realistic expectations for what products can and cannot do. Here is the honest picture, plus what actually works.
There is no permanent cure for vitiligo — but that is not the full picture. Meaningful repigmentation, long-term remission, and a dramatically improved appearance are all achievable with current treatments. Here is the honest answer.
Yes — repigmented vitiligo can depigment again after treatment stops. Understanding relapse rates, what drives recurrence, and how to maintain results with the minimum ongoing intervention.
Yes — vitiligo can stop spreading without treatment, and does so in a significant proportion of patients. Understanding what spontaneous stabilisation means, when it is likely, and how to distinguish it from temporary plateaus.
Vitiligo itself does not damage internal organs. The condition is confined to melanocytes in the skin and hair. But the autoimmune mechanism behind vitiligo is associated with other conditions that do affect internal health — and these are worth understanding.
Summer makes vitiligo more visible and can trigger new patches through sunburn and Koebner effect — but it is not categorically worse for the underlying disease. Here is how to understand the seasonal dimension of vitiligo.
Vitiligo patches themselves do not typically itch — but some patients do experience itch, and the reasons are worth understanding. Here is what the research shows.
Excimer laser therapy delivers targeted narrowband UVB to specific vitiligo patches, producing better results for limited disease than whole-body phototherapy — but at higher cost and limited availability. Here is the complete picture.
Vitiligo spreads at widely different rates — from imperceptibly slow to rapid progression over weeks. Understanding what drives spread, what rapid progression looks like, and when to act can help you respond appropriately rather than with unnecessary panic or false reassurance.
Having a ready, comfortable response to questions about vitiligo — whether from colleagues, strangers, or children — reduces anxiety and gives you control over these interactions. Here are practical scripts for different situations.
Yes — vitiligo is an autoimmune disease. Understanding what that means at a mechanistic level explains why it co-occurs with other autoimmune conditions, why JAK inhibitors work, and what treatment is actually targeting.
